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VIC is involved in various molecular mechanisms that underlie its anticarcinogenic effect

Fibrosis to Carcinogenesis: Unveiling the Causal Dynamics between Pulmonary Fibrosis and Lung Cancer

Activation of p53 by MDM2-inhibition sensitizes quiescent blast crisis CML stem/progenitor cells to Bcl-2 inhibitor- and tyrosine kinase inhibitor-induced cell death

Similarly, it is not uncommon for ostensibly compelling results, that may indeed be statistically significant, to be amplified while other relevant findings of significant consumer interest are obscured or omitted (e.g

We have recently started to look at the molecular and cellular effects of ITCs in plants [1518] in order to find the molecular mechanisms underlying some of these macroscopic observations

What would happen to astrocytes and microglia as a response to increased MGO if the BBB is already lost, for instance during neuroinflammation, is unclear